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Down-regulation of macrophage immune activity by natural CD8plus regulatory T cells

100%

Bryniarski K. , Szczepanik M. , Ptak M. , Sikora E. , Majewska-Szczepanik M. , Ptak W.

Folia Biologica

2013

tom 61

nr 1-2

A new physiological role of copper amine oxidases: cardioprotection against reactive oxygen intermediates

100%

Mateescu M. A. , Dumoulin M. J. , Wang X. T. , Nadeau R. , Mondovi B.

Journal of Physiology and Pharmacology. Supplement

1997

tom 48

nr 2

Assessment of the influence of hormonal replacement therapy in postmenopausal women on reactive oxygen intermediates generation by neutrophils

100%

Stetkiewicz T. , Polac I. , Stachowiak G. , Jedrzejczyk S. , Makula A. , Pertynski T.

Acta Toxicologica

2003

tom 11

nr 2

Plant immunology: a sleigh ride through the SNO

80%

Loake G.

BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology

2013

tom 94

nr 2

Neutrophils-induced increase of adenosine triphosphate depletion in rat neonatal cardiac myocytes with impaired energy metabolism

80%

Gajewski M. , Laskowska-Bozek H. , Maslinski S. , Ryzewski J.

Journal of Physiology and Pharmacology

1991

tom 42

nr 4

Isolated, cultured rat neonatal cardiac myocytes were placed in medium suppled menfed with mitochondrial respiratory inhibitor potassium cyanide which caused a rapid adenosine triphosphate (ATP) depletion. These myocytes with the impaired energy metabolism (“hypoxia-like state”) were exposed to unstimulated human neutrophils. Effect of human neu rophils on the myocy es m the “hypoxia-like state” was quantified as a total change in the amount of ATP in cardiac cells. After 5 hours of incubation of neu rophils with the myocytes in the “hypoxia-like state” an additional decrease (of 50 per cent) in ATP content was observed. Since catalase (which destroys hydrogen peroxide) prevented the further decline in ATP level in the myocy es with impaired energy metabolism, it seem that hydrogen peroxide and possibly their products are responsible for this effect. These results suggest that unstimulated human neu rophils af er activation by the contact with injured cardiac cells caused further decrease of ATP level in target cells.

The influence of adrenergic and cholinergic agents on the respiratory burst of human neutrophils from peripheral blood and synovial fluid

80%

Gajewski M. , Gujski M. , Ksiezopolska-Pietrzak K. , Jozwicka M. , Maslinski S.

Journal of Physiology and Pharmacology. Supplement

1997

tom 48

nr 2