Wyniki wyszukiwania - Biblioteka Nauki

1

Mathematical modeling of telomere shortening: an overview

100%

Olofsson P.

Archives of Control Sciences

|

1999

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tom Vol. 9, no. 1/2

133-141

EN

Shortening of telomeres is one of the supposed mechanisms of aging and death. This paper reviews the attempts to model and analyze this process with mathematical methods. The emphasis is put on branching process models where the main features are irreducibility and polynomial growth.

2

Tranformation of a human mixed GH-PRL adenoma cell population in response to long-term bromocriptine treatment

75%

Bacsy E. , Fazekas I. , Slowik F. , Pasztor E. , Czirjak S.

Folia Histochemica et Cytobiologica

|

1997

|

tom 35

|

nr 2

3

Apigenin inhibits growth and motility but increases gap junctional coupling intensity in rat prostate carcinoma [MAT-LyLu] cell populations

63%

Czernik M. , Sroka J. , Madeja Z. , Czyz J.

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tom 13

|

nr 3

327-338

EN

Apigenin (4′,5,7,-trihydroxyflavone) is a flavonoid abundant in the common fruits, herbs and vegetables constituting the bulk of the human diet. This study was aimed at quantifying the effects of apigenin on the basic cellular traits determining cancer development, i.e. cell proliferation, gap junctional coupling, and motility, using the Dunning rat prostate MAT-LyLu cell model. We demonstrated that apigenin considerably inhibits MAT-LyLu cell proliferation and significantly enhances the intensity of connexin43-mediated gap junctional coupling. This effect correlates with an increased abundance of C×43-positive plaques at the cell-to-cell borders seen in apigenin-treated variants. Moreover, we observed an inhibitory effect of apigenin on the motility of MAT-LyLu cells. The basic parameters characterising MAT-LyLu cell motility, especially the rate of cell displacement, considerably decreased upon apigenin administration. This in vitro data indicates that apigenin may affect cancer development in general, and prostate carcinogenesis in particular, via its influence on cellular activities decisive for both cancer promotion and progression, including cell proliferation, gap junctional coupling and cell motility and invasiveness.

4

The influence of liposomes containing cholesterol on phagocytic activity and proliferation of Tetrahymena cells

63%

Fabczak H.

Acta Protozoologica

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1988

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tom 27

|

nr 2

5

Comparison of lectin induced chemotactic selection and chemical imprinting in Tetrahymena pyriformis

63%

Kohidai L. , Banky C. , Csaba G.

Acta Protozoologica

|

2003

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tom 42

|

nr 2

6

The dimerized lysosyme [DL] effects on the peritoneal cell population influenced by lipopolysaccharides [LPS]. The cell nucleus response

63%

Lukaszyk A. , Klein P. , Gozdzicka-Jozefiak A. , Balcerek M. , Gorska-Rafinska L. , Kiczka W. , Ludwiczak H. , Piescikowska I. , Sawinski P.

Folia Histochemica et Cytobiologica. Supplement

|

1997

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tom 35

|

nr 2

7

Comparison of techniques permitting to detect apoptosis in situ

63%

Drag-Zalesinska M. , Wysocka T. , Dumanska M. , Jagoda E. , Zabel M.

Folia Histochemica et Cytobiologica

|

2002

|

tom 40

|

nr 2

8

Targeting liposomes and lipoplexes to cells in the liver

51%

Scherphof G. L. , Koning G. , Bartsch M. , Yan X. , Kamps J.

Cellular and Molecular Biology Letters

|

2002

|

tom 07

|

nr 2

9

Myonuclear accretion - a brief review

51%

Mozdziak P. E. , Giamario C. , Petitte J. N.

Animal Science Papers and Reports. Supplement

|

2003

|

tom 21

|

nr 1

10

Changes in murein composition during the cell cycle of Caulobacter crescentus

44%

Markiewicz Z. , Bielecki J.

Acta Microbiologica Polonica

|

1992

|

tom 41

|

nr 3-4