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Sedimentary characteristics of the Hüdaý Formation (Early Cambrian) within the Aydincik (ÝÇEL) area, S Turkey

100%

Eren M. , Öner F.

tom Vol. 70, No. 3-4

251-259

The study area is located in Aydýncýk (ÝÇEL) area where Early Cambrian rocks are represented by the Hüdai Formation. The formation represents three different parts in the vertical sequence which has a thickness of approximately 830 metres. The basal part of the formation consists of a rhytmic alternation of horizontal laminated sandstone, ripple trough cross-laminated sandstone, mixed sandstone-shale, and rarely metashales. The middle part comprises dominantly metashales. The upper part is made up by an alternation of horizontal laminated sandstone, mixed sandstone-shale and rarely metashales. Vertical variations in the formation is caused by marine transgressious and regressious, and by fluctuations in sediment supply. Lower to middle parts of the formation characterize a fining-upward sequence, and middle to upper parts display a coarsening-upward sequence. In the Hüdai Formation, four lithofacies are recognized based on lithology and sedimentary structures. These are (1) horizontal laminated sandstone (predominantly quartzarenite) indicating deposition from tidal currents during the high velocity phases of tidal cycles; (2) ripple cross-laminated sandstone (predominantly quartzwacke) deposited in lower part of intertidal (sand-flat) environment; (3) mixed sandstone-shale revealing deposition in mid-intertidal flat environment; and (4) shale (or metashale) deposited mainly in upper intertidal (mudflat) environment.

Changes in malondialdehyde level and catalase activity and effect of toltrazuril on these parameters in chicks infected with Eimeria tenella

80%

Eraslan G. , Cam Y. , Eren M. , Liman B. C.

nr 3

Efficacy of N-acetylcysteine on aflatoxicosis in rabbits

61%

Cam Y. , Eraslan G. , Atasever A. , Eren M. , Liman B. C. , Seybek N.

2008

tom 17

nr 2

189-197

The aim of this study was to determine the clinical, hematological, biochemical and pathological findings, and to evaluate the efficiency of n-acetylcysteine (NAC) in experimentally induced aflatoxicosis in rabbits. Our study divided 42 rabbits into 6 groups. The groups received the following treatment: Group 1: control group without treatment, Group 2: aflatoxin (AF) (0.4 mg/kg body weight (bw)), Group 3: AF (0.4 mg/kg bw) plus NAC (250 mg/kg bw), Group 4: AF (0.4 mg/kg bw) plus NAC (500 mg/kg bw), Group 5: NAC (250 mg/kg bw), Group 6: NAC (500 mg/kg bw). N-acetylcysteine was administered intramuscularly on day 1 following the administration of AF for 5 days. At the beginning of the study and on days 1, 4 and 7 following the administration of AF, blood samples were collected for haematological and biochemical analysis. Necropsy and histopathological examination were performed. Clinical signs were observed starting from day 1 following the administration of AF. The signs of toxicosis included decreased feed and water consumption, dullness, dehydration, emaciation and convulsion. Four rabbits died in Group 2 and 2 rabbits died in Groups 3 and 4 treated with NAC. During the study, white blood cell (WBC) counts, mean platelet volume (MPV), mean corpuscular volume (MCV) and red blood cell distribution width (RDW) values, serum urea levels and aspartate aminotransferase (AST), alanine aminotransferase (ALT), alkaline phosphatase (ALP) and gamma glutamyl transpeptidase (GGT) activities increased whereas red blood cell (RBC) and platelet counts, hemoglobin (Hgb) and hematocrit (Hct) values, total protein, total cholesterol, triglyceride and glucose levels decreased for Group 2 given only AF compared with controls. These parameters approximated to control levels for Groups 3 and 4 after the treatment with NAC. Histopathological examination showed that the main affected organ was liver. Lipid degeneration, destruction and fibrosis in the liver were detected for Group 2. Histopathological changes in the liver were observed to be less severe for Groups 3 and 4. The study demonstrated that administration of NAC might be useful for the treatment of aflatoxicosis in rabbits.