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Distribution of indoor radon concentrations between selected Hungarian thermal baths

100%

Shahrokhi A. , Nagy E. , Csordás A. , Somlai J. , Kovács T.

Nukleonika

2016

tom Vol. 61, No. 3

333--336

Owing to the high potential of radon to increase the risk of lung cancer, health organizations are enforced to update their regulations and recommendations regarding indoor radon levels each year. In this study, the indoor radon concentrations of three randomly selected thermal baths in Hungary using CR-39 and an AlphaGUARD radon monitor were measured with regard to the new updated standards of the European Basic Safety Standard (EU BSS, Council Directive 2013/59/Euratom, 2014). The annual average of indoor radon concentrations in Parad Medical Bath, Igal Health Spa and Eger Turkish Bath were measured as 159 ± 19, 176 ± 27 and 301 ± 30 Bq/m3, respectively. Indoor radon concentration in all measurement locations were determined to be below the reference level, with the exception of the main pool, small pool and sparkling bath areas in the Eger Turkish Bath that were measured as 403 ± 42, 315 ± 32 and 354 ± 36 Bq/m3, respectively. In light of the results, the estimated annual average radon concentration in the thermal baths was below the EU BSS reference level of 300 Bq/m3. Personal dosimetry is required to estimate the annual effective dose from inhaled radon by the workers at the Eger Turkish Bath. This procedure is required in order to justify the application of the mitigation process of decreasing working hours, improving the ventilation rate or increasing the number of classified employees in response to the official radiation surveillance programme.

Preliminary results of radon survey in thermal spas in V4 countries

58%

Műllerová M. , Mazur J. , Blahušiak P. , Grządziel D. , Holý K. , Kovács T. , Kozak K. , Nagy E. , Neznal M. , Neznal M. , Shahrokhi A.

Nukleonika

2016

tom Vol. 61, No. 3

303--306

Radon concentration was measured in 11 thermal spas in Visegrad countries (Czech Republic, Hungary, Poland, and Slovakia). The results showed that in 84% of spas radon activity concentration is less than 400 Bq·m–3. However, areas with radon activity concentration exceeding 1000 Bq·m–3 were found in the Czech Republic and Slovakia as well. Preliminary analyses indicated that the highest radon activities in spas were found in places with thermal pools. Radon concentration in waters used in spas ranged from 0.5 Bq/l to 384 Bq/l. The influence of radon activity concentration in water on radon activity in the air inside the spa was observed. It was found to increase indoor radon with increasing radon in the waters. Correlation with indoor radon and radon in water was more significant for baths and less significant for pool waters. In the cases filling of the bath from water taps, significantly contribute to the increased radon was observed in the pool and bath areas of the spa.

BDNF modifies hippocampal KCC2 and NKCC1 expression in a temporal lobe epilepsy model

58%

Eftekhari S. , Mehrabi S. , Soleimani M. , Hassanzadeh G. , Shahrokhi A. , Mostafavi H. , Hayat P. , Barati M. , Mehdizadeh H. , Rahmanzadeh R. , Mostafavi H. , Barati M. , Mehdizadeh H. , Rahmanzadeh R. , Hadjighassem M. R. , Joghataei M. T.

Acta Neurobiologiae Experimentalis

2014

tom 74

nr 3

Excitatory GABA actions, induced by altered expression of chloride transporters (KCC2/NKCC1), can contribute to seizure generation in temporal lobe epilepsy. In the present study, we evaluated whether BDNF administration can affect KCC2/ NKCC1 expression, ictogenesis and behavioral alterations in this paradigm. Status epilepticus was induced in male rats with pilocarpine, followed by a treatment of either a single high dose or multiple injections of BDNF during the latent phase of temporal lobe epilepsy. Chloride transporters expression, spontaneous recurrent seizures, and hyperexcitability post-seizural behaviors were evaluated after treatment. NKCC1 protein expression was markedly upregulated, whereas that of KCC2 was significantly downregulated in epileptic hippocampi compared to intact controls. Application of BDNF (both single high dose and multiple injections) increased KCC2 expression in epileptic hippocampi, while NKCC1 expression was downregulated exclusively by the single high dose injection of BDNF. Development of spontaneous recurrent seizures was delayed but not prevented by the treatment, and hyperexcitability behaviors were ameliorated for a short period of time. To prevent GABA-A mediated depolarization and design appropriate treatment strategies for temporal lobe epilepsy, chloride transporters can be considered as a target. Future studies are warranted to investigate any possible therapeutic effects of BDNF via altering chloride transporters expression.